[1] Sullivan H S. The onset of schizophrenia[J]. The American Journal of Psychiatry, 1927, 151(6 Suppl):134-139.
[2] Yung A R, McGorry P D. The initial prodrome in psychosis:Descrip-tive and qualitative aspects[J]. Australian and New Zealand Journal of Psychiatry, 1996, 30(5):587-599.
[3] Yung A R, Phillips L J, McGorry P D, et al. Prediction of psychosis. A step towards indicated prevention of schizophrenia[J]. The British Jour-nal of Psychiatry, 1998, 172(33):14-20.
[4] Yung A R, Yuen H P, Berger G, et al. Declining transition rate in ultra high risk (prodromal) services:Dilution or reduction of risk[J]. Schizo-phrenia Bulletin, 2007, 33(3):673-681.
[5] Fusar P P, Borgwardt S, Bechdolf A, et al. The psychosis high-risk state:A comprehensive state-of-the-art review[J]. JAMA Psychiatry, 2013, 70(1):107-120.
[6] Nelson B, Yuen H P, Wood S J, et al. Long-term follow-up of a group at ultra high risk ("prodromal") for psychosis:The PACE 400 study[J]. JAMA Psychiatry, 2013, 70(8):793-802.
[7] Zheng L N, Wang J J, Zhang T T, et al. The Chinese version of the SIPS/SOPS:A pilot study of reliability and validity[J]. Chinese Mental Health Journal, 2012, 26(8):571-576.
[8] Xu L, Zhang T, Zheng L, et al. Psychometric properties of prodromal questionnaire-brief version among Chinese help-seeking individuals[J]. PloS One, 2016, 11(2):e0148935.
[9] Zhang T H, Li H J, Woodberry K A, et al. Prodromal psychosis detec-tion in a counseling center population in China:An epidemiological and clinical study[J]. Schizophrenia Research, 2014, 152(2/3):391-399.
[10] Zhang T H, Li H J, Woodberry K A, et al. Two-year follow-up of a Chinese sample at clinical high risk for psychosis:Timeline of symp-toms, help-seeking and conversion[J]. Epidemiology and Psychiatric Sciences. 2016(1):1-12.
[11] Carrión R E, Cornblatt B A, Burton C Z, et al. Personalized prediction of psychosis:External validation of the NAPLS-2 psychosis risk calcu-lator with the EDIPPP project[J]. The American Journal of Psychiatry, 2016, 173(10):989-996.
[12] Seidman L J, Shapiro D I, Stone W S, et al. Association of neurocogni-tion with transition to psychosis. baseline functioning in the second Phase of the north American prodrome longitudinal study[J]. JAMA Psychiatry, 2016, 73(12):1239-1248.
[13] Zhang T H, Tang Y Y, Cui H R, et al. Theory of mind impairments in youth at clinical high risk of psychosis[J]. Psychiatry, 2016, 79(1):40-55.
[14] Zhang T H, Yi Z H, Li H J, et al. Faux pas recognition performance in a help-seeking population at clinical high risk of psychosis[J]. European Archives of Psychiatry and Clinical Neuroscience, 2016, 266(1):71-78.
[15] Zhang T H, Cui H R, Tang Y Y, et al. Correlation of social cognition and neurocognition on psychotic outcome:A naturalistic follow-up study of subjects with attenuated psychosis syndrome[J]. Scientific Reports, 2016(6):35017.
[16] Allen P, Luigjes J, Howes O D, et al. Transition to psychosis associat-ed with prefrontal and subcortical dysfunction in ultra high-risk indi-viduals[J]. Schizophrenia Bulletin. 2012, 38(6):1268-1276.
[17] Takahashi T, Wood S J, Yung A R, et al. Progressive gray matter re-duction of the superior temporal gyrus during transition to psychosis[J]. Archives of General Psychiatry, 2009, 66(4):366-376.
[18] Sun D, Stuart G W, Jenkinson M, et al. Brain surface contraction mapped in first-episode schizophrenia:A longitudinal magnetic reso-nance imaging study[J]. Molecular Psychiatry, 2009, 14(10):976-986.
[19] Pantelis C, Velakoulis D, McGorry P D, et al. Neuroanatomical abnor-malities before and after onset of psychosis:A cross-sectional and lon-gitudinal MRI comparison[J]. The Lancet, 2003, 361(9354):281-288.
[20] Cannon T D, Chung Y, He G, et al. Progressive reduction in cortical thickness as psychosis develops:A multisite longitudinal neuroimag-ing study of youth at elevated clinical risk[J]. Biological Psychiatry, 2015, 77(2):147-157.
[21] Koutsouleris N, Meisenzahl E M, Davatzikos C, et al. Use of neuroana-tomical pattern classification to identify subjects in at-risk mental states of psychosis and predict disease transition[J]. Archives of General Psychiatry, 2009, 66(7):700-712.
[22] Wang J J, Tang Y X, Li C B, et al. Decreased P300 current source density in drug-naive first episode schizophrenics revealed by high density recording[J]. International Journal of Psychophysiology, 2010, 75(3):249-257.
[23] Spencer K M, Salisbury D F, Shenton M E, et al. Gamma-band audito-ry steady-state responses are impaired in first episode psychosis[J]. Biological Psychiatry, 2008, 64(5):369-375.
[24] Bodatsch M, Brockhaus D A, Klosterkötter J, et al. Forecasting psycho-sis by event-related potentials-systematic review and specific metaanalysis[J]. Biological Psychiatry, 2015, 77(11):951-958.
[25] Janssen C I, Kiliaan A J. Long-chain polyunsaturated fatty acids (LCP-UFA) from genesis to senescence:The influence of LCPUFA on neu-ral development, aging, and neurodegeneration[J]. Progress in Lipid Research, 2014, 53(53):1-17.
[26] Amminger G P, Schafer M R, Klier C M, et al. Decreased nervonic ac-id levels in erythrocyte membranes predict psychosis in help-seeking ultra-high-risk individuals[J]. Molecular Psychiatry, 2012, 17(12):1150-1152.
[27] Lavoie S, Whitford T J, Benninger F, et al. Correlates of electroenceph-alographic resting states and erythrocyte membrane docosahexaenoic and eicosapentaenoic acid levels in individuals at ultra-high risk of psychosis[J]. Australian and New Zealand Journal of Psychiatry, 2016, 50(1):56-63.
[28] Amminger G P, Schafer M R, Papageorgiou K, et al. Long-chain ome-ga-3 fatty acids for indicated prevention of psychotic disorders:A ran-domized, placebo-controlled trial[J]. Archives of General Psychiatry, 2010, 67(2):146-154.
[29] McGorry P D, Nelson B, Markulev C, et al. Effect of omega-3 polyun-saturated fatty acids in young people at ultrahigh risk for psychotic disorders:The NEURAPRO randomized clinical trial[J]. JAMA Psychi-atry, 2016, doi:10.1001/jamapsychiatry.2016.2902.
[30] Addington J, Epstein I, Liu L, et al. A randomized controlled trial of cognitive behavioral therapy for individuals at clinical high risk of psy-chosis[J]. Schizophrenia Research, 2011, 125(1):54-61.
[31] Fusar P P, Frascarelli M, Valmaggia L, et al. Antidepressant, antipsy-chotic and psychological interventions in subjects at high clinical risk for psychosis:OASIS 6-year naturalistic study[J]. Psychological Medi-cine, 2015, 45(6):1327-1339.
[32] Rauchensteiner S, Kawohl W, Ozgurdal S, et al. Test-performance af-ter cognitive training in persons at risk mental state of schizophrenia and patients with schizophrenia[J]. Psychiatry Research, 2011, 185(3):334-339.
[33] Bechdolf A, Wagner M, Ruhrmann S, et al. Preventing progression to first-episode psychosis in early initial prodromal states[J]. The British Journal of Psychiatry, 2012, 200(1):22-29.
